Interactive Renogram Explorer
Drag either kidney's diuretic-renogram curve and read the phases, quantitative indices, split function, flow phase, and captopril response live
A bilateral teaching bench for the ⁹⁹ᵐTc-MAG3 / DTPA dynamic renogram. Drag either kidney's curve — its perfusion upstroke, cortical peak (Tmax), and drainage tail — and the panel decodes it live: the three phases, washout T½, output efficiency and NORA, relative (split) function, the diuretic (furosemide) response, and the O'Reilly pattern. A separate flow-phase strip and a paired ACE-inhibitor (captopril) module show how the study is actually read across its two acquisitions, and the reference gallery loads classic drainage patterns onto the curve for comparison.
Read it the way you would a real study: the curve is only one axis — always pair it with relative function and the images. A prolonged washout T½ is not diagnostic of obstruction on its own (dehydration, poor function, a capacious reservoir, and bladder backpressure all prolong it), which is why the tool foregrounds quantitative drainage indices (output efficiency, NORA) and the diuretic response rather than the raw half-time. And renovascular disease is defined by the change after the ACE-inhibitor, not the resting curve.
Bilateral renogram
drag the ● handles on the selected kidneyPhase-by-phase reading
Interpretation
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Two acquisitions, read together
The perfusion (flow) study and the dynamic renogram are separate acquisitions; renovascular disease is defined by the ACE-inhibitor change, not the resting curve.
Perfusion / flow phase
A separate acquisition — rapid 1–2 s/frame over the first ~60 s after a bolus, distinct from the 10–20 s/frame dynamic renogram above. Kidney first-pass tracks the aortic bolus; blunted or asymmetric flow is nonspecific, and focally absent perfusion is a vascular emergency (arterial/venous thrombosis; in transplants quantified by the Hilson perfusion index). Driven here by each kidney's perfusion handle.
ACE-inhibitor (captopril) renography
Baseline vs post-ACEI on the same kidney. A haemodynamically significant renal-artery stenosis relies on efferent (angiotensin-II) tone to hold GFR; the ACE-inhibitor removes it, so the post-ACEI curve deteriorates — that change, not the baseline, is the test.
Reference drainage patterns
Each mini-plot overlays the pattern (color) on a normal curve (dashed). Click to load it into the right kidney above.